Well it looks like all the drama I've experienced in regards to having such severe attacks of TN - may not just be TN after all.
My doctors are theorising at the moment, that I'm also experiencing Cluster headaches, and they may be contributing as a trigger for the TN. They then call this "Cluster-Tic Syndrome" meaning I get both TN & Cluster headaches :( these conditions are joined only by their seperate connections to the Trigeminal nerve. So at any one time, I may have one or the other of these disorders but over time they eventually layer over each other and I do now get them both at the same time....
The theory in Cluster headaches is the hypothalmus sends a signal to the arteries and veins around the trigeminal nerve, causing them to dilate (swell up) to allow more blood flow through. Because these veins are then around the trigeminal nerve, it's setting off / triggering more attacks - so I'm all at once suffering two separate yet oddly jointed disorders.
Here below is the transcript I've found on what Cluster-Tic Syndrome is in brief:
The diagnosis of cluster-tic syndrome is given to patients in whom cluster headache and trigeminal neuralgia coexist.  This rare condition is characterized by repetitive volleys of excruciating lancinating pains that usually are described with trigeminal neuralgia, but in this syndrome they occur superimposed on the typical features of cluster headache. These tic like pains commonly are precipitated by light cutaneous or mucous membrane stimulation as in typical trigeminal neuralgia. Concurrently the patient also suffers from the typical clinical features of cluster headache. Often the two pain syndromes occur independently of each other for months or years before their simultaneous occurrence. In general, the cluster-tic syndrome is difficult to treat and may require a combination of therapies. Carbamazepine has been reported to abolish the tic like pains, whereas standard anticluster agents are required to treat the features of cluster. Solomon et al described four patients with this syndrome who underwent microvascular decompression of the trigeminal nerve; compression of the root entry zone by a blood vessel was noted in all four patients. In two of the three patients in whom there was facial nerve exploration, an arterial loop was found at the root entry zone of the facial nerve. Postoperatively, all patients experienced relief of the neuralgia component of the syndrome; the features of cluster were not altered by the surgery, however.